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In the brain, as in the heart, the mitoKATP channel opener cromakalim protects neurons in vitro against both oxygen and glucose deprivation (OGD) and glutamate excitotoxicity [47]. A more mitochondrial-specific opener of KATP channels (BMS 191095) reduced infarct volume in mice subjected to Middle Cerebral Artery Occlusion (MCAO) if given 24 h previously [48]. However, the role of these channels remains unclear as there is some suggestion that blocking, rather than opening, them is protective in hippocampal slices subjected to OGD (using glibenclamide) [49] and in mice subjected to MCAO (using 5HD) [50].

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Biochem Biophys Res Commun 255: 40, 1999. 13. Bouillaud F, Couplan E, Pecqueur C, and Ricquier D. Homologues of the uncoupling protein from brown adipose tissue (UCP1): UCP2, UCP3, BMCP1 and UCP4. Biochim Biophys Acta 1504: 107, 2001. 14. Hunter DR and Haworth RA. The Ca2+-induced membrane transition in mitochondria. I. The protective mechanisms. Arch Biochem Biophys 195: 453, 1979. 15. Hunter DR and Haworth RA. The Ca2+-induced membrane transition in mitochondria. III. Transitional Ca2+ release.

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